| Download | - View final version: Can Campylobacter coli induce Guillain-Barre syndrome? (PDF, 580 KiB)
|
|---|
| DOI | Resolve DOI: https://doi.org/10.1007/s10096-008-0661-9 |
|---|
| Author | Search for: Van Belkum, A.; Search for: Jacobs, B.; Search for: van Beek, E.; Search for: Louwen, R.; Search for: Van Rijs, W.; Search for: Debruyne, L.; Search for: Gilbert, M.1; Search for: Li, J.1; Search for: Jansz, A.; Search for: Mégraud, F.; Search for: Endtz, H. |
|---|
| Affiliation | - National Research Council of Canada. NRC Institute for Biological Sciences
|
|---|
| Format | Text, Article |
|---|
| Subject | amplify fragment length polymorphism; healthy blood donor; 664H2004 strain; amplify fragment length polymorphism profile; strain GB50 |
|---|
| Abstract | Campylobacter jejuni enteritis is the most frequently identified infection preceding the Guillain-Barré syndrome (GBS) and neural damage is thought to be induced through molecular mimicry between C. jejuni lipo-oligosaccharide (LOS) and human gangliosides. It has been questioned whether or not other Campylobacter species, including C. curvus, C. upsaliensis and C. coli, could be similarly involved. This is relevant because it would imply that bacterial factors considered important in the aetiology of GBS crossed species barriers. Two prior reports have appeared where C. coli was putatively associated with a case of GBS. |
|---|
| Publication date | 2008-11-12 |
|---|
| Publisher | Springer |
|---|
| In | |
|---|
| Language | English |
|---|
| Peer reviewed | Yes |
|---|
| NPARC number | 15295297 |
|---|
| Export citation | Export as RIS |
|---|
| Report a correction | Report a correction (opens in a new tab) |
|---|
| Record identifier | 8fc3241a-8317-4843-91c7-5409f06b1321 |
|---|
| Record created | 2010-05-17 |
|---|
| Record modified | 2020-05-27 |
|---|
